Direct interaction between GluR2 and GAPDH regulates AMPAR-mediated excitotoxicity

Over-activation of AMPARs (α−amino-3-hydroxy-5-methylisoxazole-4-propionic acid subtype glutamate receptors) is implicated in excitotoxic neuronal death associated with acute brain insults, such as ischemic stroke. However, the specific molecular mechanism by which AMPARs, especially the calcium-imp...

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Autori principali: Wang, Min, Li, Shupeng, Zhang, Hongyu, Pei, Lin, Zou, Shengwei, Lee, Frank J S, Wang, Yu Tian, Liu, Fang
Natura: Artigo
Lingua:Inglês
Pubblicazione: BioMed Central 2012
Soggetti:
Research
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC3407747/
https://ncbi.nlm.nih.gov/pubmed/22537872
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1756-6606-5-13
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