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Direct interaction between GluR2 and GAPDH regulates AMPAR-mediated excitotoxicity
Over-activation of AMPARs (α−amino-3-hydroxy-5-methylisoxazole-4-propionic acid subtype glutamate receptors) is implicated in excitotoxic neuronal death associated with acute brain insults, such as ischemic stroke. However, the specific molecular mechanism by which AMPARs, especially the calcium-imp...
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| Main Authors: | , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
BioMed Central
2012
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3407747/ https://ncbi.nlm.nih.gov/pubmed/22537872 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1756-6606-5-13 |
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