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The KCNQ1-KCNE2 K(+) channel is required for adequate thyroid I(−) uptake
The KCNQ1 α subunit and the KCNE2 β subunit form a potassium channel in thyroid epithelial cells. Genetic disruption of KCNQ1-KCNE2 causes hypothyroidism in mice, resulting in cardiac hypertrophy, dwarfism, alopecia, and prenatal mortality. Here, we investigated the mechanistic requirement for KCNQ1...
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| 主要な著者: | , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Federation of American Societies for Experimental Biology
2012
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3405278/ https://ncbi.nlm.nih.gov/pubmed/22549510 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.12-206110 |
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