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Alanine or aspartic acid substitutions at serine23/24 of cardiac troponin I decrease thin filament activation, with no effect on crossbridge detachment kinetics

Ala/Asp substitutions at Ser23/24 have been employed to investigate the functional impact of cardiac troponin I (cTnI) phosphorylation by protein kinase A (PKA). Some limitations of previous studies include the use of heterologous proteins and confounding effects arising from phosphorylation of card...

תיאור מלא

שמור ב:
מידע ביבליוגרפי
Main Authors: Mamidi, Ranganath, Gollapudi, Sampath K., Mallampalli, Sri Lakshmi, Chandra, Murali
פורמט: Artigo
שפה:Inglês
יצא לאור: 2012
נושאים:
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC3405180/
https://ncbi.nlm.nih.gov/pubmed/22684024
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.abb.2012.05.024
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