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Tumor necrosis factor alpha stimulates cathepsin K and V activity via juxtacrine monocyte-endothelial cell signaling and JNK activation

Inflammation and damage promote monocyte adhesion to endothelium and cardiovascular disease (CVD). Elevated inflammation and increased monocyte-endothelial cell interactions represent the initial stages of vascular remodeling associated with a multitude of cardiovascular diseases. Cathepsins are pro...

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Main Authors: Keegan, Philip M., Wilder, Catera L., Platt, Manu O.
格式: Artigo
語言:Inglês
出版: 2012
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在線閱讀:https://ncbi.nlm.nih.gov/pmc/articles/PMC3390414/
https://ncbi.nlm.nih.gov/pubmed/22562303
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s11010-012-1320-0
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