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A Genomic Mechanism for Antagonism Between Retinoic Acid and Estrogen Signaling in Breast Cancer
Retinoic acid (RA) triggers growth-suppressive effects in tumor cells and therefore RA has and its synthetic analogs have great potential as anti-carcinogenic agent. RA effects are mediated by Retinoic Acid Receptors (RARs), which regulate gene expression in an RA-dependent manner. To define the gen...
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| Hauptverfasser: | , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
2009
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3374131/ https://ncbi.nlm.nih.gov/pubmed/19563758 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cell.2009.04.043 |
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