Mitochondria-targeted catalase reduces abnormal APP processing, amyloid β production and BACE1 in a mouse model of Alzheimer's disease: implications for neuroprotection and lifespan extension

Lignende værker

• Toxicity of Neurons Treated with Herbicides and Neuroprotection by Mitochondria-Targeted Antioxidant SS31
  af: Reddy, Tejaswini P., et al.
  Udgivet: (2011)
• Amyloid-β and Mitochondria in Aging and Alzheimer’s Disease: Implications for Synaptic Damage and Cognitive Decline
  af: Reddy, P. Hemachandra, et al.
  Udgivet: (2010)
• Dynamin-Related Protein 1 and Mitochondrial Fragmentation in Neurodegenerative Diseases
  af: Reddy, P. Hemachandra, et al.
  Udgivet: (2010)
• Abnormal mitochondrial dynamics, mitochondrial loss and mutant huntingtin oligomers in Huntington's disease: implications for selective neuronal damage
  af: Shirendeb, Ulziibat, et al.
  Udgivet: (2011)
• Impaired mitochondrial biogenesis, defective axonal transport of mitochondria, abnormal mitochondrial dynamics and synaptic degeneration in a mouse model of Alzheimer's disease
  af: Calkins, Marcus J., et al.
  Udgivet: (2011)