Deletion of Gα(Z) Protein Protects against Diet-induced Glucose Intolerance via Expansion of β-Cell Mass

Insufficient plasma insulin levels caused by deficits in both pancreatic β-cell function and mass contribute to the pathogenesis of type 2 diabetes. This loss of insulin-producing capacity is termed β-cell decompensation. Our work is focused on defining the role(s) of guanine nucleotide-binding prot...

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Bibliografische gegevens
Hoofdauteurs: Kimple, Michelle E., Moss, Jennifer B., Brar, Harpreet K., Rosa, Taylor C., Truchan, Nathan A., Pasker, Renee L., Newgard, Christopher B., Casey, Patrick J.
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: American Society for Biochemistry and Molecular Biology 2012
Onderwerpen:
Molecular Bases of Disease
Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3370216/
https://ncbi.nlm.nih.gov/pubmed/22457354
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M112.359745
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