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Pharmacological Chaperoning of Nicotinic Acetylcholine Receptors Reduces the Endoplasmic Reticulum Stress Response

We report the first observation that endoplasmic reticulum (ER) stress and the unfolded protein response (UPR) can decrease when a central nervous system drug acts as an intracellular pharmacological chaperone for its classic receptor. Transient expression of α4β2 nicotinic receptors (nAChRs) in Neu...

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書誌詳細
主要な著者: Srinivasan, Rahul, Richards, Christopher I., Xiao, Cheng, Rhee, Doreen, Pantoja, Rigo, Dougherty, Dennis A., Miwa, Julie M., Lester, Henry A.
フォーマット: Artigo
言語:Inglês
出版事項: The American Society for Pharmacology and Experimental Therapeutics 2012
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC3362896/
https://ncbi.nlm.nih.gov/pubmed/22379121
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1124/mol.112.077792
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