Prevention of posttraumatic axon sprouting by blocking CRMP2-mediated neurite outgrowth and tubulin polymerization

Epileptogenesis following traumatic brain injury (TBI) is likely due to a combination of increased excitability, disinhibition, and increased excitatory connectivity via aberrant axon sprouting. Targeting these pathways could be beneficial in the prevention and treatment of posttraumatic epilepsy. H...

Πλήρης περιγραφή

Αποθηκεύτηκε σε:
Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς: Wilson, Sarah M., Xiong, Wenhui, Wang, Yuying, Ping, Xingjie, Head, Jessica D., Brittain, Joel M., Gagare, Pravin D., Ramachandran, P. Veeraraghavan, Jin, Xiaoming, Khanna, Rajesh
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: 2012
Θέματα:
Article
Διαθέσιμο Online:https://ncbi.nlm.nih.gov/pmc/articles/PMC3358479/
https://ncbi.nlm.nih.gov/pubmed/22433297
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuroscience.2012.02.038
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