Prevention of posttraumatic axon sprouting by blocking CRMP2-mediated neurite outgrowth and tubulin polymerization

Epileptogenesis following traumatic brain injury (TBI) is likely due to a combination of increased excitability, disinhibition, and increased excitatory connectivity via aberrant axon sprouting. Targeting these pathways could be beneficial in the prevention and treatment of posttraumatic epilepsy. H...

Disgrifiad llawn

Wedi'i Gadw mewn:
Manylion Llyfryddiaeth
Prif Awduron: Wilson, Sarah M., Xiong, Wenhui, Wang, Yuying, Ping, Xingjie, Head, Jessica D., Brittain, Joel M., Gagare, Pravin D., Ramachandran, P. Veeraraghavan, Jin, Xiaoming, Khanna, Rajesh
Fformat: Artigo
Iaith:Inglês
Cyhoeddwyd: 2012
Pynciau:
Article
Mynediad Ar-lein:https://ncbi.nlm.nih.gov/pmc/articles/PMC3358479/
https://ncbi.nlm.nih.gov/pubmed/22433297
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuroscience.2012.02.038
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