GlialCAM, a Protein Defective in a Leukodystrophy, Serves as a ClC-2 Cl(−) Channel Auxiliary Subunit

Ion fluxes mediated by glial cells are required for several physiological processes such as fluid homeostasis or the maintenance of low extracellular potassium during high neuronal activity. In mice, the disruption of the Cl(−) channel ClC-2 causes fluid accumulation leading to myelin vacuolation. A...

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Wedi'i Gadw mewn:
Manylion Llyfryddiaeth
Prif Awduron: Jeworutzki, Elena, López-Hernández, Tania, Capdevila-Nortes, Xavier, Sirisi, Sònia, Bengtsson, Luiza, Montolio, Marisol, Zifarelli, Giovanni, Arnedo, Tanit, Müller, Catrin S., Schulte, Uwe, Nunes, Virginia, Martínez, Albert, Jentsch, Thomas J., Gasull, Xavier, Pusch, Michael, Estévez, Raúl
Fformat: Artigo
Iaith:Inglês
Cyhoeddwyd: Cell Press 2012
Pynciau:
Article
Mynediad Ar-lein:https://ncbi.nlm.nih.gov/pmc/articles/PMC3334819/
https://ncbi.nlm.nih.gov/pubmed/22405205
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuron.2011.12.039
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