GlialCAM, a Protein Defective in a Leukodystrophy, Serves as a ClC-2 Cl(−) Channel Auxiliary Subunit

Ion fluxes mediated by glial cells are required for several physiological processes such as fluid homeostasis or the maintenance of low extracellular potassium during high neuronal activity. In mice, the disruption of the Cl(−) channel ClC-2 causes fluid accumulation leading to myelin vacuolation. A...

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Autors principals: Jeworutzki, Elena, López-Hernández, Tania, Capdevila-Nortes, Xavier, Sirisi, Sònia, Bengtsson, Luiza, Montolio, Marisol, Zifarelli, Giovanni, Arnedo, Tanit, Müller, Catrin S., Schulte, Uwe, Nunes, Virginia, Martínez, Albert, Jentsch, Thomas J., Gasull, Xavier, Pusch, Michael, Estévez, Raúl
Format: Artigo
Idioma:Inglês
Publicat: Cell Press 2012
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Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3334819/
https://ncbi.nlm.nih.gov/pubmed/22405205
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuron.2011.12.039
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