GlialCAM, a Protein Defective in a Leukodystrophy, Serves as a ClC-2 Cl(−) Channel Auxiliary Subunit

Ion fluxes mediated by glial cells are required for several physiological processes such as fluid homeostasis or the maintenance of low extracellular potassium during high neuronal activity. In mice, the disruption of the Cl(−) channel ClC-2 causes fluid accumulation leading to myelin vacuolation. A...

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Bibliografski detalji
Glavni autori: Jeworutzki, Elena, López-Hernández, Tania, Capdevila-Nortes, Xavier, Sirisi, Sònia, Bengtsson, Luiza, Montolio, Marisol, Zifarelli, Giovanni, Arnedo, Tanit, Müller, Catrin S., Schulte, Uwe, Nunes, Virginia, Martínez, Albert, Jentsch, Thomas J., Gasull, Xavier, Pusch, Michael, Estévez, Raúl
Format: Artigo
Jezik:Inglês
Izdano: Cell Press 2012
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Article
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3334819/
https://ncbi.nlm.nih.gov/pubmed/22405205
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuron.2011.12.039
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