GlialCAM, a Protein Defective in a Leukodystrophy, Serves as a ClC-2 Cl(−) Channel Auxiliary Subunit

Ion fluxes mediated by glial cells are required for several physiological processes such as fluid homeostasis or the maintenance of low extracellular potassium during high neuronal activity. In mice, the disruption of the Cl(−) channel ClC-2 causes fluid accumulation leading to myelin vacuolation. A...

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Detalhes bibliográficos
Main Authors: Jeworutzki, Elena, López-Hernández, Tania, Capdevila-Nortes, Xavier, Sirisi, Sònia, Bengtsson, Luiza, Montolio, Marisol, Zifarelli, Giovanni, Arnedo, Tanit, Müller, Catrin S., Schulte, Uwe, Nunes, Virginia, Martínez, Albert, Jentsch, Thomas J., Gasull, Xavier, Pusch, Michael, Estévez, Raúl
Formato: Artigo
Idioma:Inglês
Publicado em: Cell Press 2012
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3334819/
https://ncbi.nlm.nih.gov/pubmed/22405205
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuron.2011.12.039
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