Ablation of junctin or triadin is associated with increased cardiac injury following ischaemia/reperfusion

AIMS: Junctin and triadin are calsequestrin-binding proteins that regulate sarcoplasmic reticulum (SR) Ca(2+) release by interacting with the ryanodine receptor. The levels of these proteins are significantly down-regulated in failing human hearts. However, the significance of such decreases is curr...

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Autors principals: Cai, Wen-Feng, Pritchard, Tracy, Florea, Stela, Lam, Chi-Kueng, Han, Peidong, Zhou, Xiaoyang, Yuan, Qunying, Lehnart, Stephan E., Allen, Paul D., Kranias, Evangelia G.
Format: Artigo
Idioma:Inglês
Publicat: Oxford University Press 2012
Matèries:
Original Articles: Spotlight on Reducing Myocardial Ischaemia/Reperfusion Injury
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3331615/
https://ncbi.nlm.nih.gov/pubmed/22411973
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/cvr/cvs119
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