Cytoskeletal defects in Bmpr2-associated pulmonary arterial hypertension

The heritable form of pulmonary arterial hypertension (PAH) is typically caused by a mutation in bone morphogenic protein receptor type 2 (BMPR2), and mice expressing Bmpr2 mutations develop PAH with features similar to human disease. BMPR2 is known to interact with the cytoskeleton, and human array...

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Detalhes bibliográficos
Main Authors: Johnson, Jennifer A., Hemnes, Anna R., Perrien, Daniel S., Schuster, Manfred, Robinson, Linda J., Gladson, Santhi, Loibner, Hans, Bai, Susan, Blackwell, Tom R., Tada, Yuji, Harral, Julie W., Talati, Megha, Lane, Kirk B., Fagan, Karen A., West, James
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2012
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Editorial Focus
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3311512/
https://ncbi.nlm.nih.gov/pubmed/22180660
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00202.2011
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