Cytoskeletal defects in Bmpr2-associated pulmonary arterial hypertension

The heritable form of pulmonary arterial hypertension (PAH) is typically caused by a mutation in bone morphogenic protein receptor type 2 (BMPR2), and mice expressing Bmpr2 mutations develop PAH with features similar to human disease. BMPR2 is known to interact with the cytoskeleton, and human array...

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Dades bibliogràfiques
Autors principals: Johnson, Jennifer A., Hemnes, Anna R., Perrien, Daniel S., Schuster, Manfred, Robinson, Linda J., Gladson, Santhi, Loibner, Hans, Bai, Susan, Blackwell, Tom R., Tada, Yuji, Harral, Julie W., Talati, Megha, Lane, Kirk B., Fagan, Karen A., West, James
Format: Artigo
Idioma:Inglês
Publicat: American Physiological Society 2012
Matèries:
Editorial Focus
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3311512/
https://ncbi.nlm.nih.gov/pubmed/22180660
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00202.2011
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