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Cytoskeletal defects in Bmpr2-associated pulmonary arterial hypertension
The heritable form of pulmonary arterial hypertension (PAH) is typically caused by a mutation in bone morphogenic protein receptor type 2 (BMPR2), and mice expressing Bmpr2 mutations develop PAH with features similar to human disease. BMPR2 is known to interact with the cytoskeleton, and human array...
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| Autors principals: | , , , , , , , , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Physiological Society
2012
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3311512/ https://ncbi.nlm.nih.gov/pubmed/22180660 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00202.2011 |
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