iNOS deficiency ameliorates skeletal muscle insulin resistance but does not alter unexpected lower blood glucose levels after burn injury in C57BL/6 mice

OBJECTIVE: Burn injury is associated with inflammatory responses and metabolic alterations including insulin resistance. Impaired insulin receptor substrate-1 (IRS-1)-mediated insulin signal transduction is a major component of insulin resistance in skeletal muscle following burn injury. To further...

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Hlavní autoři: Sugita, Michiko, Sugita, Hiroki, Kim, Minhye, Mao, Ji, Yasuda, Yoshikazu, Habiro, Mayu, Shinozaki, Shohei, Yasuhara, Shingo, Shimizu, Nobuyuki, Martyn, J.A. Jeevendra, Kaneki, Masao
Médium: Artigo
Jazyk:Inglês
Vydáno: 2011
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On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3304504/
https://ncbi.nlm.nih.gov/pubmed/21816442
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.metabol.2011.06.001
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