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Rapid conversion to high xanthine oxidase activity in viable Kupffer cells during hypoxia.

It has been widely postulated that the central mechanism of hepatic reperfusion injury involves the conversion, during ischemia, of the enzyme xanthine dehydrogenase (XDH) to its free radical-producing form, xanthine oxidase (XOD). However, this theory has been questioned because (a) XDH to XOD conv...

Ausführliche Beschreibung

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Bibliographische Detailangaben
Hauptverfasser: Wiezorek, J S, Brown, D H, Kupperman, D E, Brass, C A
Format: Artigo
Sprache:Inglês
Veröffentlicht: 1994
Schlagworte:
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC330048/
https://ncbi.nlm.nih.gov/pubmed/7989578
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