Colistin-Resistant, Lipopolysaccharide-Deficient Acinetobacter baumannii Responds to Lipopolysaccharide Loss through Increased Expression of Genes Involved in the Synthesis and Transport of Lipoproteins, Phospholipids, and Poly-β-1,6-N-Acetylglucosamine

We recently demonstrated that colistin resistance in Acinetobacter baumannii can result from mutational inactivation of genes essential for lipid A biosynthesis (Moffatt JH, et al., Antimicrob. Agents Chemother. 54:4971–4977). Consequently, strains harboring these mutations are unable to produce the...

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Detalhes bibliográficos
Main Authors: Henry, Rebekah, Vithanage, Nuwan, Harrison, Paul, Seemann, Torsten, Coutts, Scott, Moffatt, Jennifer H., Nation, Roger L., Li, Jian, Harper, Marina, Adler, Ben, Boyce, John D.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Microbiology 2012
Assuntos:
Mechanisms of Resistance
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3256090/
https://ncbi.nlm.nih.gov/pubmed/22024825
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/AAC.05191-11
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