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BACE1 elevation is involved in amyloid plaque development in the triple transgenic model of Alzheimer’s disease: Differential Aβ antibody labeling of early-onset axon terminal pathology
β-amyloid precursor protein (APP) and presenilins mutations cause early-onset familial Alzheimer’s disease (FAD). Some FAD-based mouse models produce amyloid plaques, others don’t. β-Amyloid (Aβ) deposition can manifest as compact and diffuse plaques; it is unclear why the same Aβ molecules aggregat...
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| Glavni autori: | , , , , , , , , , , |
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| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
2011
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3227764/ https://ncbi.nlm.nih.gov/pubmed/21725719 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s12640-011-9256-9 |
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