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The A53T Mutation is Key in Defining the Differences in the Aggregation Kinetics of Human and Mouse α-synuclein
Despite a 95% sequence similarity, the aggregation of human and mouse α-synuclein is remarkably different, as the human form is slower than the mouse form in forming fibrils, but is associated with Parkinson’s disease both in humans and transgenic mice. Here, the amino acid code underlying these dif...
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| Main Authors: | , , , |
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| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
2011
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3205953/ https://ncbi.nlm.nih.gov/pubmed/21721555 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1021/ja203979j |
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