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Connexin43 Modulates Cell Polarity and Directional Cell Migration by Regulating Microtubule Dynamics

Knockout mice deficient in the gap junction gene connexin43 exhibit developmental anomalies associated with abnormal neural crest, primordial germ cell, and proepicardial cell migration. These migration defects are due to a loss of directional cell movement, and are associated with abnormal actin st...

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Autors principals: Francis, Richard, Xu, Xin, Park, Hyunsoo, Wei, Chin-Jen, Chang, Stephen, Chatterjee, Bishwanath, Lo, Cecilia
Format: Artigo
Idioma:Inglês
Publicat: Public Library of Science 2011
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3194834/
https://ncbi.nlm.nih.gov/pubmed/22022608
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0026379
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