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A deletion of FGFR2 creating a chimeric IIIb/IIIc exon in a child with Apert syndrome
BACKGROUND: Signalling by fibroblast growth factor receptor type 2 (FGFR2) normally involves a tissue-specific alternative splice choice between two exons (IIIb and IIIc), which generates two receptor isoforms (FGFR2b and FGFR2c respectively) with differing repertoires of FGF-binding specificity. He...
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| Hlavní autoři: | , , , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
BioMed Central
2011
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3192734/ https://ncbi.nlm.nih.gov/pubmed/21943124 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1471-2350-12-122 |
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