Angiotensin II Contributes to Podocyte Injury by Increasing TRPC6 Expression via an NFAT-Mediated Positive Feedback Signaling Pathway

The transient receptor potential channel C6 (TRPC6) is a slit diaphragm–associated protein in podocytes involved in regulating glomerular filter function. Gain-of-function mutations in TRPC6 cause hereditary focal segmental glomerulosclerosis (FSGS), and several human acquired proteinuric diseases s...

पूर्ण विवरण

में बचाया:
ग्रंथसूची विवरण
मुख्य लेखकों: Nijenhuis, Tom, Sloan, Alexis J., Hoenderop, Joost G.J., Flesche, Jan, van Goor, Harry, Kistler, Andreas D., Bakker, Marinka, Bindels, Rene J.M., de Boer, Rudolf A., Möller, Clemens C., Hamming, Inge, Navis, Gerjan, Wetzels, Jack F.M., Berden, Jo H.M., Reiser, Jochen, Faul, Christian, van der Vlag, Johan
स्वरूप: Artigo
भाषा:Inglês
प्रकाशित: American Society for Investigative Pathology 2011
विषय:
Regular Article
ऑनलाइन पहुंच:https://ncbi.nlm.nih.gov/pmc/articles/PMC3181349/
https://ncbi.nlm.nih.gov/pubmed/21839714
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ajpath.2011.06.033
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