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Loss of p53 Ser18 and Atm Results in Embryonic Lethality without Cooperation in Tumorigenesis

Phosphorylation at murine Serine 18 (human Serine 15) is a critical regulatory process for the tumor suppressor function of p53. p53Ser18 residue is a substrate for ataxia-telangiectasia mutated (ATM) and ATM-related (ATR) protein kinases. Studies of mice with a germ-line mutation that replaces Ser1...

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Sonraí Bibleagrafaíochta
Main Authors: Armata, Heather L., Shroff, Punita, Garlick, David E., Penta, Krista, Tapper, Andrew R., Sluss, Hayla K.
Formáid: Artigo
Teanga:Inglês
Foilsithe: Public Library of Science 2011
Ábhair:
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC3181255/
https://ncbi.nlm.nih.gov/pubmed/21980358
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0024813
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