NOD2 Signaling Contributes to the Innate Immune Response Against Helper-Dependent Adenovirus Vectors Independently of MyD88 In Vivo

We previously demonstrated that Toll-like receptor/myeloid differentiation primary response gene 88 (MyD88) signaling is required for maximal innate and acquired [T helper cell type 1 (Th1)] immune responses following systemic administration of helper-dependent adenoviral vectors (HDAds). However, M...

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Autors principals: Suzuki, Masataka, Cela, Racel, Bertin, Terry K., Sule, Gautam, Cerullo, Vincenzo, Rodgers, John R., Lee, Brendan
Format: Artigo
Idioma:Inglês
Publicat: Mary Ann Liebert, Inc. 2011
Matèries:
Research Articles
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3177951/
https://ncbi.nlm.nih.gov/pubmed/21561248
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/hum.2011.002
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