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Occupancy of both sites on the thyrotropin (TSH) receptor dimer is necessary for phosphoinositide signaling

The thyroid-stimulating hormone (TSH) receptor signals via G(s) to produce cAMP and via G(q/11) to produce inositol-1,4,5-trisphosphate, which is degraded to inositol monophosphate (IP1; phosphoinositide signaling). The potency of TSH for cAMP signaling is higher than for phosphoinositide signaling,...

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Detalhes bibliográficos
Main Authors: Allen, Michael D., Neumann, Susanne, Gershengorn, Marvin C.
Formato: Artigo
Idioma:Inglês
Publicado em: Federation of American Societies for Experimental Biology 2011
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3177577/
https://ncbi.nlm.nih.gov/pubmed/21705666
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.11-188961
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