Mislocalization of Large ARF-GEFs as a Potential Mechanism for BFA Resistance in COG-Deficient Cells

Defects in subunits of the conserved oligomeric Golgi (COG) complex represent a growing subset of congenital disorders of glycosylation (CDGs). In addition to altered protein glycosylation and vesicular trafficking, Cog-deficient patient fibroblasts exhibit a striking delay in the Golgi-disrupting e...

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Autores principales: Flanagan-Steet, Heather, Johnson, Steven, Smith, Richard D., Bangiyeva, Julia, Lupashin, Vladimir, Steet, Richard
Formato: Artigo
Lenguaje:Inglês
Publicado: 2011
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Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC3159704/
https://ncbi.nlm.nih.gov/pubmed/21722633
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yexcr.2011.06.005
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