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Mislocalization of Large ARF-GEFs as a Potential Mechanism for BFA Resistance in COG-Deficient Cells
Defects in subunits of the conserved oligomeric Golgi (COG) complex represent a growing subset of congenital disorders of glycosylation (CDGs). In addition to altered protein glycosylation and vesicular trafficking, Cog-deficient patient fibroblasts exhibit a striking delay in the Golgi-disrupting e...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2011
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3159704/ https://ncbi.nlm.nih.gov/pubmed/21722633 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yexcr.2011.06.005 |
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