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c-Met recruits ICAM-1 as a coreceptor to compensate for the loss of CD44 in Cd44 null mice
CD44 isoforms act as coreceptors for the receptor tyrosine kinases c-Met and VEGFR-2. However, Cd44 knockout mice do not show overt phenotypes, in contrast to Met and Vegfr-2 knockout mice. We hypothesized that CD44 is being compensated for by another factor in Cd44 null mice. Using RNAi technology...
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| Main Authors: | , , , , , , , |
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| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
The American Society for Cell Biology
2011
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3145552/ https://ncbi.nlm.nih.gov/pubmed/21680714 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1091/mbc.E11-02-0134 |
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