14-3-3theta Protects against Neurotoxicity in a Cellular Parkinson's Disease Model through Inhibition of the Apoptotic Factor Bax

Disruption of 14-3-3 function by alpha-synuclein has been implicated in Parkinson's disease. As 14-3-3s are important regulators of cell death pathways, disruption of 14-3-3s could result in the release of pro-apoptotic factors, such as Bax. We have previously shown that overexpression of 14-3-...

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Detaylı Bibliyografya
Asıl Yazarlar: Slone, Sunny R., Lesort, Mathieu, Yacoubian, Talene A.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Public Library of Science 2011
Konular:
Research Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC3140482/
https://ncbi.nlm.nih.gov/pubmed/21799745
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0021720
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