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CCR4 Contributes to the Pathogenesis of Experimental Autoimmune Encephalomyelitis by Regulating Inflammatory Macrophage Function
Chemokines and their receptors play a critical role in orchestrating the immune response during experimental autoimmune encephalomyelitis (EAE). Expression of CCR4 and its ligand CCL22 has been observed in ongoing disease. Here we describe a role for CCR4 in EAE, illustrating delayed and decreased d...
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| Hoofdauteurs: | , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
2011
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3139027/ https://ncbi.nlm.nih.gov/pubmed/21575994 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jneuroim.2011.04.008 |
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