Inhibition of growth of Chlamydia trachomatis by tumor necrosis factor is accompanied by increased prostaglandin synthesis.

Development of Chlamydia trachomatis (L2/434/Bu) in HEp-2 cells was inhibited by treatment of the cells with recombinant human alpha tumor necrosis factor (TNF). In the infected cultures that were treated with TNF, high concentrations of prostaglandin E2(PGE2) were detected, exceeding by far the con...

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Detalhes bibliográficos

Publicado no:	Infect Immun
Principais autores:	Holtmann, H, Shemer-Avni, Y, Wessel, K, Sarov, I, Wallach, D
Formato:	Artigo
Idioma:	Inglês
Publicado em:	American Society for Microbiology (ASM) 1990
Assuntos:	Research Article
Acesso em linha:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC313634/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2119339/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/iai.58.10.3168-3172.1990
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