Inhibition of growth of Chlamydia trachomatis by tumor necrosis factor is accompanied by increased prostaglandin synthesis.

Development of Chlamydia trachomatis (L2/434/Bu) in HEp-2 cells was inhibited by treatment of the cells with recombinant human alpha tumor necrosis factor (TNF). In the infected cultures that were treated with TNF, high concentrations of prostaglandin E2(PGE2) were detected, exceeding by far the con...

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Bibliografski detalji

Izdano u:	Infect Immun
Glavni autori:	Holtmann, H, Shemer-Avni, Y, Wessel, K, Sarov, I, Wallach, D
Format:	Artigo
Jezik:	Inglês
Izdano:	American Society for Microbiology (ASM) 1990
Teme:	Research Article
Online pristup:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC313634/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2119339/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/iai.58.10.3168-3172.1990
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