Inhibition of growth of Chlamydia trachomatis by tumor necrosis factor is accompanied by increased prostaglandin synthesis.

Development of Chlamydia trachomatis (L2/434/Bu) in HEp-2 cells was inhibited by treatment of the cells with recombinant human alpha tumor necrosis factor (TNF). In the infected cultures that were treated with TNF, high concentrations of prostaglandin E2(PGE2) were detected, exceeding by far the con...

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發表在:	Infect Immun
Principais autores:	Holtmann, H, Shemer-Avni, Y, Wessel, K, Sarov, I, Wallach, D
格式:	Artigo
語言:	Inglês
出版:	American Society for Microbiology (ASM) 1990
主題:	Research Article
在線閱讀:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC313634/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2119339/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/iai.58.10.3168-3172.1990
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