TWEAK and Fn14 expression in the pathogenesis of joint inflammation and bone erosion in rheumatoid arthritis

INTRODUCTION: TNF-like weak inducer of apoptosis (TWEAK) has been proposed as a mediator of inflammation and bone erosion in rheumatoid arthritis (RA). This study aimed to investigate TWEAK and TWEAK receptor (Fn14) expression in synovial tissue from patients with active and inactive rheumatoid arth...

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Hlavní autoři: Dharmapatni, Anak ASSK, Smith, Malcolm D, Crotti, Tania N, Holding, Christopher A, Vincent, Cristina, Weedon, Helen M, Zannettino, Andrew CW, Zheng, Timothy S, Findlay, David M, Atkins, Gerald J, Haynes, David R
Médium: Artigo
Jazyk:Inglês
Vydáno: BioMed Central 2011
Témata:
Research Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3132040/
https://ncbi.nlm.nih.gov/pubmed/21435232
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/ar3294
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