TWEAK and Fn14 expression in the pathogenesis of joint inflammation and bone erosion in rheumatoid arthritis

INTRODUCTION: TNF-like weak inducer of apoptosis (TWEAK) has been proposed as a mediator of inflammation and bone erosion in rheumatoid arthritis (RA). This study aimed to investigate TWEAK and TWEAK receptor (Fn14) expression in synovial tissue from patients with active and inactive rheumatoid arth...

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Tallennettuna:
Bibliografiset tiedot
Päätekijät: Dharmapatni, Anak ASSK, Smith, Malcolm D, Crotti, Tania N, Holding, Christopher A, Vincent, Cristina, Weedon, Helen M, Zannettino, Andrew CW, Zheng, Timothy S, Findlay, David M, Atkins, Gerald J, Haynes, David R
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: BioMed Central 2011
Aiheet:
Research Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3132040/
https://ncbi.nlm.nih.gov/pubmed/21435232
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/ar3294
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