TWEAK and Fn14 expression in the pathogenesis of joint inflammation and bone erosion in rheumatoid arthritis

INTRODUCTION: TNF-like weak inducer of apoptosis (TWEAK) has been proposed as a mediator of inflammation and bone erosion in rheumatoid arthritis (RA). This study aimed to investigate TWEAK and TWEAK receptor (Fn14) expression in synovial tissue from patients with active and inactive rheumatoid arth...

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Main Authors: Dharmapatni, Anak ASSK, Smith, Malcolm D, Crotti, Tania N, Holding, Christopher A, Vincent, Cristina, Weedon, Helen M, Zannettino, Andrew CW, Zheng, Timothy S, Findlay, David M, Atkins, Gerald J, Haynes, David R
Formáid: Artigo
Teanga:Inglês
Foilsithe: BioMed Central 2011
Ábhair:
Research Article
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC3132040/
https://ncbi.nlm.nih.gov/pubmed/21435232
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/ar3294
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