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TAK1 regulates SCF expression to modulate PKBα activity that protects keratinocytes from ROS-induced apoptosis
Dysregulated reactive oxygen species (ROS) generation contributes to many human pathologies, including cancer and diabetes. During normal wound repair, inflammation-induced ROS production must be tightly controlled, but the mechanisms reining their generation remain unclear. Herein, we show that tra...
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| Autors principals: | , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Nature Publishing Group
2011
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3131962/ https://ncbi.nlm.nih.gov/pubmed/21233843 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cdd.2010.182 |
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