SOCS1 deficiency results in accelerated mammary gland development and rescues lactation in prolactin receptor–deficient mice
Prolactin is essential for proliferation and differentiation of the developing mammary gland. We have explored a role for Suppressor of Cytokine Signaling 1 (SOCS1) as a modulator of the prolactin response using mice deficient in SOCS1, which were rescued from neonatal death by deletion of the Inter...
保存先:
| 出版年: | Genes Dev |
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| 主要な著者: | , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Cold Spring Harbor Laboratory Press
2001
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC312725/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11445538/ https://ncbi.nlm.nih.govhttps://doi.org/10.1101/gad.880801 |
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