Erythropoietin prevents neuronal apoptosis after cerebral ischemia and metabolic stress

Erythropoietin (EPO) promotes neuronal survival after hypoxia and other metabolic insults by largely unknown mechanisms. Apoptosis and necrosis have been proposed as mechanisms of cellular demise, and either could be the target of actions of EPO. This study evaluates whether antiapoptotic mechanisms...

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Vydáno v:	Proc Natl Acad Sci U S A
Hlavní autoři:	Sirén, Anna-Leena, Fratelli, Maddalena, Brines, Michael, Goemans, Christoph, Casagrande, Simona, Lewczuk, Piotr, Keenan, Sonja, Gleiter, Christoph, Pasquali, Claudio, Capobianco, Annalisa, Mennini, Tiziana, Heumann, Rolf, Cerami, Anthony, Ehrenreich, Hannelore, Ghezzi, Pietro
Médium:	Artigo
Jazyk:	Inglês
Vydáno:	National Academy of Sciences 2001
Témata:	Biological Sciences
On-line přístup:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC31176/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11259643/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.051606598
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