Erythropoietin prevents neuronal apoptosis after cerebral ischemia and metabolic stress

Erythropoietin (EPO) promotes neuronal survival after hypoxia and other metabolic insults by largely unknown mechanisms. Apoptosis and necrosis have been proposed as mechanisms of cellular demise, and either could be the target of actions of EPO. This study evaluates whether antiapoptotic mechanisms...

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Detalhes bibliográficos
Main Authors: Sirén, Anna-Leena, Fratelli, Maddalena, Brines, Michael, Goemans, Christoph, Casagrande, Simona, Lewczuk, Piotr, Keenan, Sonja, Gleiter, Christoph, Pasquali, Claudio, Capobianco, Annalisa, Mennini, Tiziana, Heumann, Rolf, Cerami, Anthony, Ehrenreich, Hannelore, Ghezzi, Pietro
Formato: Artigo
Idioma:Inglês
Publicado em: The National Academy of Sciences 2001
Assuntos:
Biological Sciences
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC31176/
https://ncbi.nlm.nih.gov/pubmed/11259643
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.051606598
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