Erythropoietin prevents neuronal apoptosis after cerebral ischemia and metabolic stress
Erythropoietin (EPO) promotes neuronal survival after hypoxia and other metabolic insults by largely unknown mechanisms. Apoptosis and necrosis have been proposed as mechanisms of cellular demise, and either could be the target of actions of EPO. This study evaluates whether antiapoptotic mechanisms...
Guardado en:
| Publicado en: | Proc Natl Acad Sci U S A |
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| Autores principales: | , , , , , , , , , , , , , , |
| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
National Academy of Sciences
2001
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC31176/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11259643/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.051606598 |
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