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Erythropoietin prevents neuronal apoptosis after cerebral ischemia and metabolic stress

Erythropoietin (EPO) promotes neuronal survival after hypoxia and other metabolic insults by largely unknown mechanisms. Apoptosis and necrosis have been proposed as mechanisms of cellular demise, and either could be the target of actions of EPO. This study evaluates whether antiapoptotic mechanisms...

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Publicado en:Proc Natl Acad Sci U S A
Autores principales: Sirén, Anna-Leena, Fratelli, Maddalena, Brines, Michael, Goemans, Christoph, Casagrande, Simona, Lewczuk, Piotr, Keenan, Sonja, Gleiter, Christoph, Pasquali, Claudio, Capobianco, Annalisa, Mennini, Tiziana, Heumann, Rolf, Cerami, Anthony, Ehrenreich, Hannelore, Ghezzi, Pietro
Formato: Artigo
Lenguaje:Inglês
Publicado: National Academy of Sciences 2001
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Acceso en línea:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC31176/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11259643/
https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.051606598
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