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IL-13Rα1 Expression on β-Cell–Specific T Cells in NOD Mice
OBJECTIVE: Immunotherapy using peptides from the β-cell antigen GAD65 can preserve glucose homeostasis in diabetes-prone NOD mice; however, the precise mechanisms that arrest islet-reactive T cells remain unresolved. Our previous work revealed that a dominant GAD65 epitope contained two overlapping...
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主要な著者: | , , , , |
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フォーマット: | Artigo |
言語: | Inglês |
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American Diabetes Association
2011
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オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3114389/ https://ncbi.nlm.nih.gov/pubmed/21617187 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2337/db10-1229 |
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