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Cell-specific roles of GRK2 in onset and severity of hypoxic-ischemic brain damage in neonatal mice
The ubiquitously expressed kinase GRK2 protects against cellular overstimulation by desensitizing G protein-coupled receptors and regulating intracellular signaling. Recently, we described that hypoxia-ischemia (HI)-induced brain damage was accelerated and increased in GRK2(+/−) neonatal mice. Using...
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| Main Authors: | , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2009
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3099598/ https://ncbi.nlm.nih.gov/pubmed/19932746 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbi.2009.11.009 |
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