Low Endogenous G-Protein-Coupled Receptor Kinase 2 Sensitizes the Immature Brain to Hypoxia–Ischemia-Induced Gray and White Matter Damage

Lignende værker

• The endogenous regenerative capacity of the damaged newborn brain: boosting neurogenesis with mesenchymal stem cell treatment
  af: Donega, Vanessa, et al.
  Udgivet: (2013)
• Pharmacological Neuroprotection after Perinatal Hypoxic-Ischemic Brain Injury
  af: Fan, Xiyong, et al.
  Udgivet: (2010)
• Cell-specific roles of GRK2 in onset and severity of hypoxic-ischemic brain damage in neonatal mice
  af: Nijboer, Cora H., et al.
  Udgivet: (2009)
• Repeated Mesenchymal Stem Cell Treatment after Neonatal Hypoxia–Ischemia Has Distinct Effects on Formation and Maturation of New Neurons and Oligodendrocytes Leading to Restoration of Damage, Corticospinal Motor Tract Activity, and Sensorimotor Function
  af: van Velthoven, Cindy T. J., et al.
  Udgivet: (2010)
• Combined fetal inflammation and postnatal hypoxia causes myelin deficits and autism‐like behavior in a rat model of diffuse white matter injury
  af: van Tilborg, Erik, et al.
  Udgivet: (2017)