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Regulation of L-type inward calcium channel activity by captopril and angiotensin II via the phosphatidyl inositol 3-kinase pathway in cardiomyocytes from volume-overload hypertrophied rat hearts

Heart failure can be caused by pro-hypertrophic humoral factors such as angiotensin II (Ang II), which regulates protein kinase activities. The intermingled responses of these kinases lead to the early compensated cardiac hypertrophy, but later to the uncompensated phase of heart failure. We have sh...

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Hlavní autoři: Alvin, Zikiar, Laurence, Graham G., Coleman, Bernell R, Zhao, Aiqiu, Hajj-Moussa, Majd, Haddad, Georges E.
Médium: Artigo
Jazyk:Inglês
Vydáno: 2011
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3086391/
https://ncbi.nlm.nih.gov/pubmed/21423294
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1139/Y11-011
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