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Rad51 Overexpression Rescues Radiation Resistance in BRCA2-Defective Cancer Cells
Breast cancers with BRCA2 mutations exhibit DNA repair defects and are particularly sensitive to radiation. BRCA2 interacts with Rad51 in a complex manner involving internal BRC and C-terminal TR2 domains which play a key role in homologous recombination. BRCA2 expression also modulates Rad51 protei...
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| Main Authors: | , |
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| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
2009
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3080251/ https://ncbi.nlm.nih.gov/pubmed/18618591 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/mc.20463 |
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