Lack of Chemokine Signaling through CXCR5 Causes Increased Mortality, Ventricular Dilatation and Deranged Matrix during Cardiac Pressure Overload

RATIONALE: Inflammatory mechanisms have been suggested to play a role in the development of heart failure (HF), but a role for chemokines is largely unknown. Based on their role in inflammation and matrix remodeling in other tissues, we hypothesized that CXCL13 and CXCR5 could be involved in cardiac...

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Main Authors: Waehre, Anne, Halvorsen, Bente, Yndestad, Arne, Husberg, Cathrine, Sjaastad, Ivar, Nygård, Ståle, Dahl, Christen P., Ahmed, M. Shakil, Finsen, Alexandra V., Reims, Henrik, Louch, William E., Hilfiker-Kleiner, Denise, Vinge, Leif E., Roald, Borghild, Attramadal, Håvard, Lipp, Martin, Gullestad, Lars, Aukrust, Pål, Christensen, Geir
Formáid: Artigo
Teanga:Inglês
Foilsithe: Public Library of Science 2011
Ábhair:
Research Article
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC3078912/
https://ncbi.nlm.nih.gov/pubmed/21533157
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0018668
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