Lack of Chemokine Signaling through CXCR5 Causes Increased Mortality, Ventricular Dilatation and Deranged Matrix during Cardiac Pressure Overload

RATIONALE: Inflammatory mechanisms have been suggested to play a role in the development of heart failure (HF), but a role for chemokines is largely unknown. Based on their role in inflammation and matrix remodeling in other tissues, we hypothesized that CXCL13 and CXCR5 could be involved in cardiac...

Descripció completa

Guardat en:
Dades bibliogràfiques
Autors principals: Waehre, Anne, Halvorsen, Bente, Yndestad, Arne, Husberg, Cathrine, Sjaastad, Ivar, Nygård, Ståle, Dahl, Christen P., Ahmed, M. Shakil, Finsen, Alexandra V., Reims, Henrik, Louch, William E., Hilfiker-Kleiner, Denise, Vinge, Leif E., Roald, Borghild, Attramadal, Håvard, Lipp, Martin, Gullestad, Lars, Aukrust, Pål, Christensen, Geir
Format: Artigo
Idioma:Inglês
Publicat: Public Library of Science 2011
Matèries:
Research Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3078912/
https://ncbi.nlm.nih.gov/pubmed/21533157
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0018668
Etiquetes: Afegir etiqueta
Sense etiquetes, Sigues el primer a etiquetar aquest registre!

Ítems similars