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Interferon regulatory factor 3 and Type I interferons are protective in alcoholic liver injury in mice via cross-talk of parenchymal and myeloid cells
Alcoholic liver disease (ALD) features increased hepatic exposure to bacterial lipopolysaccharide (LPS). Toll-like receptor-4 (TLR4) recognizes LPS and activates signaling pathways depending on MyD88 or TRIF adaptors. We previously showed that MyD88 is dispensable in ALD. TLR4 induces Type-I interfe...
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| Glavni autori: | , , , , , , , , , |
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| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
2011
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3069538/ https://ncbi.nlm.nih.gov/pubmed/21274885 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/hep.24059 |
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